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New methods reveal cancer mechanism in ancient genes
Fred Hutch researchers discover that overproduction of DNA packaging material predicts aggressive brain and breast tumors, which could lead to cheaper diagnostic tests and new drug therapies
Benign meningiomas can’t stop YAP-ing, but aggressive subtypes are more soft-spoken
From the Holland Lab, Human Biology Division
Oncogenic role of a developmentally regulated NTRK2 splice variant
From the Holland Lab, Human Biology Division
Innovative Holland Lab mouse model helps tease apart YAP1’s role in brain cancer
From the Holland Lab, Cancer Basic Biology Program, Cancer Consortium.
The NTRK2 splice variant TrkB.T1 plays a vital role in human glioma
From the Holland lab, Human Biology Division
Macrophages can be the heroes too against glioblastoma
From the Holland lab, Human Biology Division
Anticipation of VEGF-mediated resistance to oncolytic viruses in glioblastoma
From the Holland lab, Human Biology Division
Arming oncolytic viruses to fight glioblastoma
From the Holland lab, Human Biology Division
Clinical trials for glioblastoma may be missing a common and aggressive molecular subtype
From the Holland lab, Human Biology Division
NF-κB is not the only player in C11orf95-RELA fusion driven tumors
From the Holland lab, Human Biology Division
Brain cancer subtype caused by single gene fusion
Hutch researchers create genetically relevant model to examine the biology of supratentorial ependymomas
Getting unlucky with chromosome number 7
From the Holland lab, Human Biology Division